ALS or MOTOR NEURON DISEASE CAUSED BY LYME DISEASE

Dr. Al Miller Lyme Disease Intro
Differential Diagnosis of Lyme & Borreliosis

Published on Apr 19, 2017

This is the introduction to my 4 part series on Lyme Disease.

For more information contact me at lymediagnosis@gmail.com

The above was shared by a friend Dana Parish with the following information -'Iwas surprised and delighted to receive a call from retired Mayo Clinic rheumatologist, Dr. Alfred Miller, last week. He wanted to tell me about his daughter-in-law, also named Dana, who was struck by ALS at age 43. He could not imagine this happening to this beautiful, healthy woman and started investigating. 

Upon researching, he discovered links between Lyme and ALS and had her tested. WHAT DO YOU KNOW!!! She was POSITIVE! Remember, though, he had her tested at a proper lab, IGeneX, and NOT Quest or Lab Corp, which miss approx 50% of cases!

So, all the "best" doctors in the world sent her home to die with no hope and no proper investigation into the CAUSE of her ALS. Those of us in the Lyme community are well aware that the Mayo Clinic is the LAST place you want to go if you have Lyme.

Dr. Miller began treating her aggressively with IV antibiotics and they halted the progression of her illness. This does not happen with "real" ALS. 

He began contacting his patients who he had previously diagnosed with Fibromyalgia, Rheumatoid arthritis, and other inflammatory "auto-immune" arthritis and diseases and urged them to get properly assessed for Lyme and other tick-borne diseases known to cause these symptoms, like Bartonella. 

WHAT DO YOU KNOW!! Most of them had Lyme!!

I look forward to bringing you more from Dr. Miller soon. In the meantime, he is on a mission to educate doctors and patients about the ravages of this disease and his horror at the medical community's ignorance about it.

All Dr Miller's presentations are available on You Tube  

https://www.youtube.com/channel/UCD19kTsVBMH-F0BfV3335Ow 

Thank you Dr Miller for having an open mind, after a lifetime in Rheumatology you were still willing to learn and seek out answers, but more importantly thank you for speaking up and sharing your experiences.

Singer/songwriter Dana Parish was involved in the recent Fox5NY News 

LYME & REASON which I posted about https://lookingatlyme.blogspot.co.uk/2016/07/fox-5-lyme-and-reason.html 

Thrilled to share that FOX 5 / Fox5NY.com Lyme and Reason Special won the Emmy for Best Science/ Health program, as presented by Dr. Oz.

MOTOR NEURON DISEASE/ALS STABILIZED ON LONG TERM ANTIBIOTICS

Sharing from a friend with a link to a post on my blog about him here

The translation is thanks to Google so apologise for errors but you get the idea.

Dear Joanne 

would you please translate this to English? Its a column to have been underwritten wooden i will send to ILADS guys! borreliasyk I've been in 12 years. 

Like many others, I have not been believed that the Norwegian health care system that diagnosed me with ALS. I've driven trial twice. Lost both. This despite the fact that I tested positive on the Western Blot test, tested positive at the Vestfold Hospital and found Borrelia DNA in my blood in 2009. 

I sometimes have a feeling of being involuntary participant in an academic parlor game. It's as if most of the health care system sees this as a game - and not as a prolonged, intense and bitter struggle for life and health. , we have a large group of seriously ill patients who have good reason to believe that a Borrelia infection is the underlying cause - but are banned from using the device with a negative antibody test (often Elisa) as justification. 

In 12 years I have accumulated knowledge about the disease, but it seems that doctors have a monopoly on qualified opinions. Because of their status, they can decide what is the scope for credible evidence to justify saying their opinions. 

If they encounter documentation that goes across their opinions, they may with ease doubt about it. Or so they know not to studies and publications that make their arguments fall through. The latter is probably hard to recognize, so it will be easier to call documentation implausible. 

I refer to the magazine's story about how Borrelia among others Dr. Jon Sundal attended. He was also an expert witness against me in the trial. I'm not looking for the actual content of the program, but there are general attitudes Sundal have, I want to life! 

One tries to persuade public opinion that good western blot tests often give false positive responses, which is incorrect tests based on several numerical markers that identify Borrelia bacterium. Some markers are nonspecific and may cross react with other bacteria, while some markers are specific borrelia "fingerprint" - markers. Mon. require a certain number of markers to be regarded as positive for declaring the test as positive. 

This will make it easier to evaluate the tests. requirement to have a positive test, three of these specific markers. 

The American Health Authority (CDC) has recognized my test as secure positive. micrograms recombinant western blot (immunoblot) containing antigen that captures all strains of Borrelia bacteria. It does not Elisa! . Recommended Vestfold Hospital (SIV's) MIKROGEN recombinant western blot that is the best test.

 The problem is again money. The test is expensive and time consuming. This must be funded! SIV has expertise and a highly skilled specialists! 

Sundal also claims that one must have inflammatory cells in the spinal fluid of having neuroborreliosis. This Slovenian researcher Franck Strle proven wrong. He is with Hunfeld Fingerle Wilske and Stanek among tungtvektekterne in Europa.Også laboratories in Norway have discovered this! Unfortunately, doctors are not updated for this No, absolutely no Norwegian attending their seminars! Only a retired doctor, Per Bjarke. 

False negative tests dominate. Therefore, it is dangerous to allude that Elisa tests are good enough. Lori Bakken conducted a double-blind study showed that not only was Elisa inaccuracies between competing laboratories, but the same laboratory showed different results on the same sample. Only 45 laboratories had a score of 55 percent.

 Another study was conducted by the College of American Patholigists. The result was terrible. It was similar to the same number of false positives as false negatives. Only 45 percent of the 516 laboratories that tested showed the correct answer. It entered that stage I of the disease discovers Elisa 20-50 percent of cases and in stage II / III 70-100 percent, depending on the tests used. 

Some argue that sensitivity in stage II / III is not more than 20-80 per cent. The tests used in the cerebrospinal fluid in Norway today finds no more than 50 percent: 70 percent for garinii and 10 percent for afzelii, which is the most common borrelia strain circulating in Norway due to false negative results are numerous. 

The bacteria manipulate our immune system so that we do not produce antibodies. Antibodies are very late in forføpet. The bacteria can lie in niches inside the cells of the organs where it is hidden from the immune system. The bacterium converted to cyst forms with new antigens that are not found in the tests available today. 

The tests used today are quite specific as false positive is not a big problem. False negatives, however, problematic. This is why clinic and patients' own history is so important in the overall assessment. I think Western blot should be emphasized rather than as indirect Elisa test. The sensitivity has been discussed regarding western blot, but specific band in the test should be a clear indicator of Borrelia infection. 

ANTIBIOTICS For chronically ill have a pulsating regime (periodic time) high doses of ceftriaxone IV has been a success. This was published in the early 90's and was confirmed on MLDA Lyme congress in 2002. treatment should be individualized on both the dosage and length. 

Stricker refers to studies where long-term antibiotic treatment is far better than the standard treatment. Klempners standard (Dr Mark Klem opens conducted a study on the treatment of patients. Though the study had clear deficiencies, it has been accepted as standard. Probably why we have several cases of illness due under treatment) in a month ceftriaxone IV and two months of oral doxycycklin, had little effect on chronic borrelia or come late disease. 

Increased improvement the longer treatment duration was noticeable. This is published in the CID that is a IDSA (Infectious Disease Society of America). Looks like IDSA turns when they choose to publish this. So in these cases must be treated for months and even years with antibiotics. 

Change of combination treatments may be necessary. Must be individualized. It is vital to acknowledge this since late come and chronic Borrelia ill probably have chronic infection combined with additional infections. Therefore, required prolonged treatment. Therefore I genuinely sorry when doctors refuse patients treatment! There is no reason to doubt that prolonged treatment helps some. 

According to statistics I should have been dead 10 years ago! I'm probably the only one in the world who has lived with ALS diagnosis in 12 years, which can stand on their feet and do not need a respirator! Court, the appeal board patient and their experts find it more probable than that I have borrelia. 

Fortunately rescued antibiotics I received in the USA for 03 my life! I had to go. Here at home did not help! Health care is often concerned with the ethical dimension. As I see it, it is the exclusion of Lyme disease patients is the greatest ethical challenge. These patients (assuming they indisputably have very severe disease) are invited ME diagnosis or ALS without other evidence than that it can not be anything else. 

It may NOT be ethically problematic to make treatment studies in this group of patients. They get anyway no qualified help. I will fight for us, as long as there is life in me.

Eivind Markhus

TREATMENT TO REVERSE MOTOR NEURON DISEASE.

'Any therapy which is capable of reversing the untreated

natural history of Motor Neuron Disease is Press-worthy.'

inmacdonald

Posts: 548

Joined: Fri 13 Jan 2012 22:32

by inmacdonald » Fri 23 Nov 2012 17:22

ALS-like Lyme and ALS not related to Lyme Neuroborreliosis:

Let us reduce the discussion to progressive incapacitating Motor Neuron Disease.
Dr William Harvey ,now deceased from a Heart Attack in year 2012 an buried with honor at the Cemetery of the US Air force Academy, developed Motor Neuron disease,progressive type.
He diagnosed himself ..He started a course of Long term antiborrelia antiBabesial antimicrobialtherapy.
Dr. William Harvey experienced a reversal of his motor neuron disease after long term antibiotic therapy.

Dr David Martz developed progressive motor neuron disease. He was diagnosed by several consultant neurologists as "most consistent with Amotropic Lateral Sclerosis. His children searched the Internet
and found the newspaper reports of Dr William Harvey's results with the success of long term antibiotic
and antibabesia therapy in motor neuron disease. Dr David Martz became a patient of Dr Bill Harvey.
Long term Therapy with antiborrelia/antibabesial medications produced a reversal of his disease. but from time to time, Dr David Martz must restart the Dr Harvey Protocol to maintain his recovery.

Dr. David Martz and I had a conversation on Nov 3, 2012 in Boston. We discussed our "MarkTwain" moments. Dr.Martz has since his recovery, treated multiple patients with Antibiotics/antibabesial medications and has noted reversal of motor neuron disease in his patients. He is drafting a manuscript
to report these patients. It is likely that the completed manuscript will be published in a Scandinavian Neurology Journal, just as his case report was published. It is noteworthy that previous to the acceptance of
the Harvey Martz manuscript in a Scandinavian Journal, it was reviewed and rejected by multiple journals based in the USA and in the United Kingdom.

Any therapy which is capable of reversing the untreated natural history of Motor Neuron Disease is Press-worthy. The untreated natural history of Motor Neuron Disease is a short pathway to death by suffocation.
Above all , do no harm.
Best, 
Alan B> MacDonald MD

Motor neuron disease recovery associated with IV 
ceftriaxone and anti-Babesia therapy

Conclusion

We have documented the full neurological recovery in a patient 
with an unrecoverable MND. Thesuccessful clinical outcome was 
associated with antibiotic therapy in response to evidence of two 
concurrent infections. 

We suggest that MND may be associated with an infectious trigger
in certain cases. The use of antibiotic therapy in MND merits further evaluation.

The above comments by Dr MacDonald was posted on 
Lymenet Forum here  in response to my comments about 
an ALS/MND forum post.

Earlier posts about Dr Martz and his research can be found here  

SUCCESSFUL TREATMENT OF MOTOR NEURONES DISEASE, ALS, LOU GEHRIG'S

Dr. David Martz -- unedited footage from Bev Feldman on Vimeo.

Dr Martz was diagnosed with Motor Neurones Disease ( Amyotrophic Lateral Sclerosis, ALS, Lou Gehrig's Disease) in 2003.

Above he talks about his journey in being tested for Lyme Disease but repeated negative serology until eventually a positive DNA test in Urine.

He was treated with aggressive IV antibiotics, he says the change was dramatic.

'Within 1 month the body pain that had required oxycodone had disappeared, the arthritis that had required Intramuscular Methatroxate had totally disappeared.

Within 1 month my energy went from half an hour to with 4 to 5 hours stamina.

By 2 months I was able to cross my legs without using my arms to assist them.

By 3 months I could get up out of a chair myself without anyone having to pull me up.

By 6 months my Neurological findings had disappeared.

I did not return to absolute complete normality but did return to about 70% of my base line self and was subsequently able to open a new practice to see if my response to antibiotics might be shared by others stricken down with the usually fatal diagnosis of ALS ( MND). 

Rocky Mountain Chronic Disease Specialists was opened in 2005 and we saw approximately 900 patients over the next 2 1/2 years.

 About 100 of them had ALS ( MND) we did identify that some patients did respond to antibiotic therapy regardless of the positivity or negativity of their Lyme Disease testing by any laboratories.

We found about 15% of people clearly got functionally better and probably 20- 30% had their disease stabilized, without progressing further.

This is unheard of and we are in the process of preparing this information for publication.'

________________________________

An earlier post on Dr Martz here

PROTOMYXZOA RHEUMATICA IMPLICATED IN CHRONIC ILLNESSES

Listen to internet radio with Focus on Health on Blog Talk Radio

1 Step Blood Test Discovers Protozoa under Biofilm Structure

by Focus on Health

in Health

Fri, May 20, 2011 Dr Stephen Fry Interview

Thanks to Better Health Guy for the following  here

Steve Fry MD

• Presented on FL1953 which is now called Protomyxzoa rheumatica.
• Has found Ivermectin, Flagyl, and a low-fat diet to be helpful.
• Studies that have been done on Multiple Sclerosis patients show that patients live longer when on low-fat diets.
• Likes testing from Infectolab from Dr. Armin Schwarzbach for Chlamydia and Mycoplasma.
• Biofilm is an aggregate of microorganisms in which cells adhere to each other or to a surface.  Bacterial biofilms are a structured community of bacterial cells enclosed in a self-produced matrix according to Bill Costerton, a world leader in biofilm research.
• Lipids (fats) play a role in biofilms.
• Biofilms impact teeth, drinking water, paper manufacturing, ship hulls, medical implants, food processing, cooling towers, oil recovery, and much more.
• Iron, calcium, and magnesium all play a role in biofilm formation; be careful about the minerals that you use as these may add to the biofilms.
• There are 1,000 organisms in normal oral flora found in biofilms.
• Biofilms consist of extracellular DNA, proteins, and polysaccharides.
• Biofilms are microbial cells and EPS (extracellular polymeric substances).  EPS may be 50-90% of the biolfilm.
• Microbes are quorum sensing – biofilm communities talk to each other.  Decision making is made by decentralized groups to coordinate behavior.  This is used to coordinate gene expression.
• When microbes come together in a group, it becomes a more complex entity.
• Biofilms play a role in many diseases including ear/nose/throat, dental, respiratory, urology, orthopedic, chronic wounds, medical devices, catheters, chronic inflammation and osteonecrosis.
• Many areas of disease in the body are related to biofilms.  Coronary artery disease, MS, ALS all have association with biofilm communities.
• 46/50 children with otitis media (ear infection) had biofilms.  Chronic rhinosinusitis is a combination of  biofilms and several microbes.
• Biofilm plays a role in cystic fibrosis.
• Nanobacteria can be a cause for kidney stones and generates biofilm.
• In chronic wounds, bacteria is protected from systemic antibiotics and host defenses by biofilms which makes infection difficult to clear.
• Actinomyces, Acinetobacter, Treponema, and others are found in dental biofilms.
• Pseudomonas aeruginosa is a bacteria that is a profound biofilm former.
• Biofilm infections are difficult to eradicate.
• The immune system recognizes the infection, but it cannot eradicate it.
• Items that have been researched in biofilm treatment: Manuka honey, enzymes, multiple antibiotics, bismuth thiols, restricting metals, botanicals, mechanical removal, and EDTA to help chelate magnesium.   Magnesium is a main stabilizing force in biofilms.
• Other substances that are of interest to biofilm researchers include: Lactoferrin, Xylitol, Gallium, Dispersin, Farnesol, RNAIII inhibiting peptide, and Furanone C30.
• Corneal eye disease may be Acanthamoeba infection (protozoan).
• Protomyxzoa rheumatica (FL1953) is an Apicomplexa.
• CCSVI is a very hot topic but is also quite controversial.  One has to suspect biofilm communities.
• Organisms living in biofilm communities are usually not culturable.
• Biofilms are the rule, not exception.  They are ubiquitous.
• Book – "Biofilm Primer" by Bill Costerton.
• There is a Center for Biofilm Engineering at Montana State University - http://www.biofilm.montana.edu/
• Silver is a well known biofilm inhibitor.
• Ozone is used in industry to reduce biofilms.  Not sure if it works in humans, but may break up biofilm communities.
• Fry Labs does microscopy, serology, and molecular diagnostics.
• Protomyxzoa is an inflammatory trigger and vascular pathogen.
• Louis Pasteur believed that all diseases are caused by infections.
• Some autoimmune conditions include Graves, Hashimoto’s, Insulin-Dependent Diabetes Mellitus, Insulin Requiring Diabetes Mellitus, Multiple Sclerosis, Myasthenia Gravis, ALS, Systemic Lupus Erythematosus, Rheumatoid Arthritis, Sjogren's.
• Thomas McPherson Brown suggested that autoimmune disease is infectious in nature; primarily Mycoplasma.  The book “The Arthritis Breakthrough” was written by Brown in 1992.
• Fry has seen Protomyxzoa and biofilm in CFS, Fibromyalgia, Scleroderma, Rheumatoid Arthritis, Lupus, Multiple Sclerosis, ALS, Parkinson’s, Autism, and other conditions.
• In their smear testing, they originally identified Hemobartonella which included Hemoplasma. Today, they suggest that Epierythrocytic bacteria is a better term.  They rarely find actual Bartonella when doing PCR testing but have found cousins.
• Even a single organism can protect itself with biofilm.
• People with parasitic infections are immunocompromised.
• They have found a host of different types of bacteria in biofilms – Ralstonia, Acinetobacter (commonly seen in many patients).
• Protomyxzoa is a slime forming protozoa.  It produces biofilm.
• In ALS, 6 of 6 tested had Protomyxzoa.  5 of 6 tested has Ralstonia which is also a biofilm former.  They see profound biofilm communities.
• When one is infected with Plasmodium as a child, it reduces later incidence of Multiple Sclerosis.  The efficacy of quinine in the treatment of MS supports this connection.
• They are able to culture Protomyxzoa now.  The entire thing becomes like gelatin and they cannot get it off the microtiter plates.
• They have seen filaments as long as 3 or 4 inches in some blood samples.  When sticky stuff is coming out during a blood draw, it may not be a clot, but may be a filament or biofilm.
• In CCSVI, filaments of Protomyxzoa may be involved.  It is a vascular disease and may lead to inflammation of the vessel or vasculitis.
• Protomyxzoa has been isolated from Culex tarsalis and Culex quinquefasciatus mosquitoes.  81% by PCR carried Protomyxzoa.
• Dogs have Protomyxzoa more than cats and the older the dog, the more likely they are to have Protomyxzoa.
• Diseases are infectious and a biofilm forming protozoan could be at the heart of the problem.
• Treatment may include Tetracyclines, Plaquenil, Flagyl, herbs, enzymes, McDougall diet, and mechanical interventions such as CCSVI venoplasty.
• One should generally not consume arginine, folic acid, or magnesium as these may strengthen the protozoan and thus the biofilm.
• As for folic acid supplementation, some breads have 5% folic acid by weight.  Folic acid may increase cancer risk.  Protozoans love folic acid.
• For autoimmune conditions, antimalarials, antibiotics, anti-protozoals, anti-fungals, anti-biofilm agents, biologics, and dietary modifications may be beneficial.
• Enbrel may be helpful in some for reducing the inflammatory response.
• Fry Labs is now working on drug sensitivity studies.
• CCSVI treatment includes a mechanical clearing or balloon procedure.  It runs about 10K.  There was reportedly one death from a CCSVI procedure.
• Stents do not do very well in veins.
• From the Hubbard Study, about 1/3 feel worse, 1/3 feel nothing, and 1/3 get better with some significantly better.  One bedridden runner was running again after the procedure.
• Protozoans love lipids (fats).  The McDougall diet is used as part of treatment.  Doxycycline and tetracyclines may target the fatty acid synthesis machinery.
• Toxoplasmosis is also dependent on fats.
• Protomyxzoa grows 100 times faster with fats than without.
• There is a reduction in relative biofilms with the McDougall diet.
• In some people where they had seen the organism and biofilms, they could not find the organism after being on the McDougall diet.  Unfortunately, after starting to eat higher fat content, the microbe was again present and visible.
• They did a test in people with Protomyxzoa using a 12.5 day water fast and levels of Protomyxzoa dropped to undetectable.  Within 2 days of eating again, it was back.
• Protomyxzoa is found in CFS, Fibromyalgia, Rheumatoid Arthritis, Lupus, Crohn’s, MS, Parkinson’s, ALS, Autism, Scleroderma, and others.
• Protomyxzoa is Public Enemy #1.
• Protomyxzoa loves fat.  It is complex.  It is drug-resistant.
• Antiprotozoals or anthelmintics may be good options.
• Roy Swank Diet – 0 people on a regular diet lived with MS for 30 years.  In those on low fat diets, all but 1 was still alive.
• Protomyxzoa leads to vascular disease and chronic inflammation.  There are coagulation impairments and retrograde venous blood flow.
• Many systemic diseases can be explained as vascular phenomenon.
• Protomyxzoa is likely transmitted by mosquitoes.  Ticks are being analyzed.
• The protozoan is believed to be the foundation pathogen.
• It is not an intracellular bug; it is a big bug.  Likely exists more on the surface of the RBC, not inside.
• Patients often get worse when on magnesium.  He does not support the use of topical magnesium either.
• Bismuth may play a role as a biofilm inhibitor.
• Omega fats from chicken and plants are probably the best.
• Interestingly, it may be that magnesium is sequestered in the body and not always low as magnesium levels seem to go back to normal when the Protomyxzoa is treated.
• Dr. Fry previously had a success rate of over 70% with his patients.  Since introducing dietary modifications, this has now gone to over 90%.
• http://www.frylabs.com

For information about the work of Dr Brown which Dr Fry refers to see The Roadback Foundation here  and an earlier post here  with a video of Dr Brown and his work.

MULTIPLE SCLEROSIS AND LYME DISEASE?

Local Lyme Disease Researcher to Speak in Hermantown

For the past 20 years since he was diagnosed with a MS-like disorder and treated for Lyme disease, Duluth resident Tom Grier has taken a special interest in Lyme patients that present with symptoms similar to Multiple Sclerosis.

Mr. Grier’s interest in investigating his own disease goes far beyond what the average patient would do.

You see Mr. Grier created an organization that registers symptomatic patients for tissue collection and brain autopsies at the time of their death.

His main interest is to find formerly treated Lyme patients that have gone on to be diagnosed with either MS or dementia.

“Since 1975 when Lyme disease was first described in the medical literature, it has always been an assumption that the organism that causes Lyme disease mostly an arthritic disease and easily eradicated with the traditional and current treatment protocols of antibiotics.”

Explains Mr. Grier. “The truth is no serology test or spinal fluid test can accurately detect the sequestered infection within the human brain.

The only way to know for sure is to do brain autopsies, and look directly at the brain tissue with special dyes and stains.

Without special immune-antibody stains developed by the NIH, the bacteria would remain completely invisible under the microscope.’’

“Sadly no one is doing this kind of Lyme research; oddly the medical community seems to be strangely resistant to this kind of medical research and I’d like to know why? ” asks Mr. Grier.

“Pathology is far more definitive than assumptions, and much of our current understanding of Lyme is based on a very flimsy foundation of facts most of which turned out to be completely wrong.”

Something that Mr Grier points out in his talk are the ten facts about Lyme that the experts got wrong.

As examples, Mr. Grier cites that shortly after Lyme disease was first described in 1975 but before we knew what actually caused Lyme in 1981; that the public was told many things as absolute facts about Lyme disease that all turned out to be untrue.

Grier continues, “We were told by the experts of the time that Lyme was only transmitted by a new species of tick found in the NE USA (Ixodes dammini discovered by Andrew Spielman of Harvard) so Lyme was supposed to be a regional and isolated illness.

We were told it was mainly an arthritic disease and it turns out it can cause severe neurological damage.

We were told it was not transmitted transplacentally but several fetal autopsies have dispelled that myth.

We were told by some Lyme experts that the Lyme disease rash has to be the size of a basketball or it isn’t Lyme disease.

The truth is many Lyme rashes are only a few centimeters or not even seen.

We were told that the Lyme organism isn’t an intracellular organism which can help infections hide and remain dormant and safe from the immune system.

But as it turns out Lyme disease most definitely is an intracellular disease of the brain and we have local brain autopsies that prove this to be true despite their being treated aggressively with antibiotics.”

“The basis of our work is the idea that the bacteria enters the brain early in the infection and is trapped in brain tissue and even trapped inside individual brain neurons.

This happens after the protective barrier called the blood brain barrier is broken down by the infection in the first two weeks before the Lyme tests can even detect the infection.

Then after the infection has been cleared from the blood stream either by our immune system or by antibiotics, the blood brain barrier reseals itself weeks later trapping the infection within the brain.”

Mr. Grier explains that the end result is that the immune system stops making antibodies that the Lyme tests are looking for.

All our Lyme tests are indirect tests and have many many pitfalls.

The infection in the brain remains relatively silent for years or even decades until it results in an MS-like condition.

Explains Mr. Grier, “It isn’t a matter of if this happens because we already have individual pathologies that reveal this to be true.

Following these patients for years doing frequent brain MRIs reveals long-term treatment results in a shrinking of the white-matter lesions that look similar to MS lesions in the brain.

The question is how often is it occurring and what kind of treatment strategies do we need to create to detect brain involvement earlier, and how do we best treat longstanding spirochetal infections within the human brain?”

One of the reasons Mr. Grier has decided to do this talk in Hermantown was because of a Lyme disease documentary being filmed in Twig MN by local Duluth videographer Ben Barneveld who uncovered numerous disabled patients in the local area with a history of Lyme disease.

Unexpectedly a large percentage of these treated Lyme patients progressed on after treatment to having such conditions as:

MS, ALS, Parkinson’s, Rheumatoid Arthritis, and enlarged hearts.

Most of these patients only became aware that their neighbors were also sick like themselves, after local Lyme patients organized a showing of a Lyme disease documentary at the Twig town hall.

Now several of those patients from the Twig-Hermantown area have since registered for brain autopsies and have authorized their remains for Lyme disease research.

The lecture is titled: “Lyme On The Brain” and is a talk based almost entirely on autopsy data and pathology.

Mr. Grier’s lecture is

FREE and open  to the general public

Saturday, April 28th

10 AM

Grace Lutheran Church

5600 Miller Trunk Highway

HERMANTOWN, MINN.

All questions should be directed to

donatebrain@gmail.com

Tom Grier 218-728-3914

2000 (Poland): Lyme borreliosis and Multiple sclerosis: Any Connection? PDF here 

 

A Seroepidemic study. Ann Agric Environ Med. issue 7, 141-143

Synopsis: 

10 out of 26 MS patients tested positive for Lyme borreliosis. Notes how it is virtually impossible to make a distinction between late stage Lyme disease and Multiple sclerosis, not even with MRI. Diagnosis of MS vs. late stage neuroborreliosis are guesswork – there are no reliable tests for either. Conclusion: Multiple sclerosis may often be associated with Borrelia infection.

Ann Agric Environ Med. 2000;7(2):141-3.

Lyme borreliosis and multiple sclerosis: any connection? A seroepidemic study.

Chmielewska-Badora J, Cisak E, Dutkiewicz J.

Source

Department of Occupational Biohazards, Institute of Agricultural Medicine, Jaczewskiego 2, 20-090 Lublin, Poland.

Abstract

A total of 769 adult neurological patients hospitalised in clinics and hospitals situated in the Lublin region (eastern Poland) were examined during the years 1997-2000 with ELISA test for the presence of anti-Borrelia burgdorferi sensu lato antibodies. A statististically significant (p=0.0422) relationship was found between the clinically confirmed diagnosis of multiple sclerosis and the positive serologic reaction with Borrelia antigen. Ten out 26 patients with multiple sclerosis (38.5%) showed positive serologic reaction to Borrelia, whereas among the total number of examined neurological patients the frequency of positive findings was twice as low (19.4%). The result suggests that multiple sclerosis may be often associated with Borrelia infection

PMID:

 

11153045

 

[PubMed - indexed for MEDLINE] 

Free full text

'What is interesting here Adam is that they were not looking for MS, they gave ELISA tests to 769 neurologically impaired patients over a 

3 year period and the ones that tested positive for Lyme was only 26 patients but 1/3 of the time they were  MS patients from that group of 769

That is a high correlation considering the 50 % failure rate of ELISA tests.'